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Publication Year: 2022

431 to 440 of 1,545 Results

Fig-4-CX-722_Group_PG+.fcs Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis FCS Data - 4.8 MB - MD5: fe348ead2033966dea39ba751d226b09 Fig 4. Raw data of flow cytometry, files saved in the same path were form one group (one replication).
Fig-4-CX-722_Group_unstai.fcs Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis FCS Data - 773.6 KB - MD5: d7031d5dd37bfa93485254c199f1659f Fig 4. Raw data of flow cytometry, files saved in the same path were form one group (one replication).
Fig-4-IL-1beta-TNF-alpha-qpcr.csv Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis Comma Separated Values - 3.6 KB - MD5: ab9a22e550949a9138b3ccdc6d6f2345 Fig 4. The q-PCR results of IL-1β and TNF-α expression in relative to β-actin in P. gingivalis stimulated macrophages.
Fig-4-IL-6-ELISA-no-stattic.csv Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis Comma Separated Values - 2.1 KB - MD5: 04caab2a23a2ce04ca163ac9370f304d Fig 4. The ELISA results of IL-6 secretion in P. gingivalis stimulated macrophages without the stimulation of Stattic.
Fig-4-IL-6-ELISA-with-stattic.csv Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis Comma Separated Values - 1.5 KB - MD5: 95bf4e4f4aa2ab367c7446916106f583 Fig 4. The ELISA results of IL-6 secretion in P. gingivalis stimulated macrophages with the stimulation of Stattic.
Fig-4-IL-6-STAT3-qpcr.csv Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis Comma Separated Values - 4.6 KB - MD5: 7934ecd404724fd7915e91488a29f625 Fig 4. The q-PCR results of IL-6 and Stat3 expression in relative to β-actin in P. gingivalis stimulated macrophages with or without the presence of Stattic.
Fig-5-HE-Project_CMC 200X_ch00.tif Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis TIFF Image - 7.9 MB - MD5: 7acbc250fc412793023953cf55a99a47 Fig 5. Capture of the HE staining images.
Fig-5-HE-Project_CMC 50X_ch00.tif Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis TIFF Image - 7.9 MB - MD5: ace92dd737bdc2de9844e23e270c2eac Fig 5. Capture of the HE staining images.
Fig-5-HE-Project_CON 200X_ch00.tif Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis TIFF Image - 6.7 MB - MD5: 047e21c90d785877dba95b5d74061139 Fig 5. Capture of the HE staining images.
Fig-5-HE-Project_CON 50X_ch00.tif Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis TIFF Image - 7.5 MB - MD5: aa2367e83c1a950333474a284fe5b69c Fig 5. Capture of the HE staining images.

Fig-4-CX-722_Group_PG+.fcs

Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis

FCS Data - 4.8 MB -

Fig 4. Raw data of flow cytometry, files saved in the same path were form one group (one replication).

Fig-4-CX-722_Group_unstai.fcs

Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis

FCS Data - 773.6 KB -

Fig 4. Raw data of flow cytometry, files saved in the same path were form one group (one replication).

Fig-4-IL-1beta-TNF-alpha-qpcr.csv

Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis

Comma Separated Values - 3.6 KB -

Fig 4. The q-PCR results of IL-1β and TNF-α expression in relative to β-actin in P. gingivalis stimulated macrophages.

Fig-4-IL-6-ELISA-no-stattic.csv

Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis

Comma Separated Values - 2.1 KB -

Fig 4. The ELISA results of IL-6 secretion in P. gingivalis stimulated macrophages without the stimulation of Stattic.

Fig-4-IL-6-ELISA-with-stattic.csv

Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis

Comma Separated Values - 1.5 KB -

Fig 4. The ELISA results of IL-6 secretion in P. gingivalis stimulated macrophages with the stimulation of Stattic.

Fig-4-IL-6-STAT3-qpcr.csv

Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis

Comma Separated Values - 4.6 KB -

Fig 4. The q-PCR results of IL-6 and Stat3 expression in relative to β-actin in P. gingivalis stimulated macrophages with or without the presence of Stattic.

Fig-5-HE-Project_CMC 200X_ch00.tif

Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis

TIFF Image - 7.9 MB -

Fig 5. Capture of the HE staining images.

Fig-5-HE-Project_CMC 50X_ch00.tif

Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis

TIFF Image - 7.9 MB -

Fig 5. Capture of the HE staining images.

Fig-5-HE-Project_CON 200X_ch00.tif

Nov 3, 2022 - Macrophage M1 polarization mediated via the IL-6/STAT3 pathway contributes to apical periodontitis induced by Porphyromonas gingivalis

TIFF Image - 6.7 MB -

Fig 5. Capture of the HE staining images.